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Hepatic Insufficiency: In 12 patients with mild-to-moderate hepatic dysfunction (Child-Pugh score ?10), no effect on mean Cabergoline C max or area under the plasma concentration curve (AUC) was observed. However, patients with severe insufficiency (Child-Pugh score >10) show a substantial increase in the mean Cabergoline C max and AUC, and thus necessitate caution. Elderly: Effect of age on the pharmacokinetics of Cabergoline has not been studied. In 12 healthy adult volunteers, food did not alter Cabergoline kinetics. Dose response with inhibition of plasma prolactin, onset of maximal effect, and duration of effect has been documented following single Cabergoline doses to healthy volunteers (0. In volunteers, prolactin inhibition was evident at doses >0. Higher doses produce prolactin suppression in a greater proportion of subjects and with an earlier onset and longer duration of action. In 12 healthy volunteers, 0. In hyperprolactinemic patients (n=51), the maximal prolactin decrease after a 0. The time to maximal effect was shorter for bromocriptine than Cabergoline (6 hours vs. In 72 healthy volunteers, single or multiple doses (up to 2 mg) of Cabergoline resulted in selective inhibition of prolactin with no apparent effect on other anterior pituitary hormones (GH, FSH, LH, ACTH, and TSH) or cortisol. Indications and Usage for Cabergoline. Cabergoline Tablets, USP are indicated for the treatment of hyperprolactinemic disorders, either idiopathic or due to pituitary adenomas. Cabergoline Tablets, USP are contraindicated in patients with: Uncontrolled hypertension or known hypersensitivity to ergot derivatives. History of cardiac valvular disorders, as suggested by anatomical evidence of valvulopathy of any valve, determined by pre-treatment evaluation including echocardiographic demonstration of valve leaflet thickening, valve restriction, or mixed valve restriction-stenosis. 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